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  • Search Condition : Filter (MeSH = Granulosa Cell Tumor / metabolism*)
Species Resource Title
Human and Animal Cells KGN(RCB1154) Estradiol promotes cell survival and induces Greb1 expression in granulosa cell tumors of the ovary through an ERα-dependent mechanism.
Human and Animal Cells KGN(RCB1154) microRNA-194 is increased in polycystic ovary syndrome granulosa cell and induce KGN cells apoptosis by direct targeting heparin-binding EGF-like growth factor.
Human and Animal Cells KGN(RCB1154) Transcriptomic analysis of gene cascades involved in protein kinase A and C signaling in the KGN line of human ovarian granulosa tumor cells†.
Human and Animal Cells KGN(RCB1154) RUNX3 Promotes the Tumorigenic Phenotype in KGN, a Human Granulosa Cell Tumor-Derived Cell Line.
Human and Animal Cells KGN(RCB1154) Molecular manipulation of keratin 8/18 intermediate filaments: modulators of FAS-mediated death signaling in human ovarian granulosa tumor cells.
Human and Animal Cells KGN(RCB1154) Persistent endocrine-disrupting chemicals found in human follicular fluid stimulate IGF1 secretion by adult ovarian granulosa cell tumor spheroids and thereby increase proliferation of non-cancer ovarian granulosa cells.
Human and Animal Cells KGN(RCB1154) Disruption of 17β-estradiol secretion by persistent organic pollutants present in human follicular fluid is dependent on the potential of ovarian granulosa tumor cell lines to metabolize estrogen.
Human and Animal Cells KGN(RCB1154) Inhibitor of apoptosis proteins are potential targets for treatment of granulosa cell tumors - implications from studies in KGN.
Human and Animal Cells KGN(RCB1154) The NADPH oxidase 4 is a major source of hydrogen peroxide in human granulosa-lutein and granulosa tumor cells.
Human and Animal Cells KGN(RCB1154) YAP regulates cell proliferation, migration, and steroidogenesis in adult granulosa cell tumors.
Human and Animal Cells KGN(RCB1154) Transforming growth factor alpha (TGFα) regulates granulosa cell tumor (GCT) cell proliferation and migration through activation of multiple pathways.