• 14 Hits
  • 検索条件 : 絞込み (MeSH = Retinoblastoma Protein / metabolism*)
生物種 リソース名 タイトル
ヒト・動物細胞 NIH3T3 , MCF7(RCB1904) MicroRNA-140 mediates RB tumor suppressor function to control stem cell-like activity through interleukin-6.
ヒト・動物細胞 RAW 264(RCB0535) , THP-1(RCB1189) , MCF7(RCB1904) Retinoblastoma Inactivation Induces a Protumoral Microenvironment via Enhanced CCL2 Secretion.
ヒト・動物細胞 TFK-1(RCB2537) Aspartate beta-hydroxylase promotes cholangiocarcinoma progression by modulating RB1 phosphorylation.
ヒト・動物細胞 MCF7(RCB1904) The RB-IL-6 axis controls self-renewal and endocrine therapy resistance by fine-tuning mitochondrial activity.
ヒト・動物細胞 CACO-2(RCB0988) Phosphorylated retinoblastoma protein is a potential predictive marker of irinotecan efficacy for colorectal cancer.
遺伝子材料 AxCANCre (RDB01748) The CDKN1B-RB1-E2F1 pathway protects mouse spermatogonial stem cells from genomic damage.
ヒト・動物細胞 ECC4(RCB0982) Low-penetrant RB allele in small-cell cancer shows geldanamycin instability and discordant expression with mutant ras.
ヒト・動物細胞 MC3T3-E1(RCB1126) Chloride-dependent acceleration of cell cycle via modulation of Rb and cdc2 in osteoblastic cells.
ヒト・動物細胞 WI-38(RCB0702) Modification of the Rb-binding domain of replication-competent adenoviral vector enhances cytotoxicity against human esophageal cancers via NF-kappaB activity.
ヒト・動物細胞 Wi-38 Inhibition of cyclin D1 expression and phosphorylation of retinoblastoma protein by phosmidosine, a nucleotide antibiotic.
ヒト・動物細胞 DT40(RCB1464) , Vav3^(-) DT40(Vav3^(-) DT40) , PLC-γ2^(-) DT40(RCB1469) Rac1/p21-activated kinase pathway controls retinoblastoma protein phosphorylation and E2F transcription factor activation in B lymphocytes.
実験動物マウス RBRC01361 Undifferentiated State Induced by Rb-p53 Double Inactivation in Mouse Thyroid Neuroendocrine Cells and Embryonic Fibroblasts.
線虫 tm2359 C. elegans orthologs of components of the RB tumor suppressor complex have distinct pro-apoptotic functions.
線虫 tm1489 , tm235 C. elegans ISWI and NURF301 antagonize an Rb-like pathway in the determination of multiple cell fates.