1. Home
  2. List
Reference Patent

Reference - List

  • 13 Hits
  • Search Condition : Filter (MeSH = Fertility / genetics*)
Species Resource Title
C.elegans tm6369 Loss of SET1/COMPASS methyltransferase activity reduces lifespan and fertility in Caenorhabditis elegans.
Drosophila DGRC#124205 , DGRC#126282 Large Drosophila germline piRNA clusters are evolutionarily labile and dispensable for transposon regulation.
Drosophila DGRC#104055 RDC complex executes a dynamic piRNA program during Drosophila spermatogenesis to safeguard male fertility.
Wheat KU-8939 , KU-S-4309 , KU-6-1 , KU-6-2 , KU-20-10 , KU-7856 , KU-5786 , KU-4-1 , KU-5-2 , KU-14655 Unleashing floret fertility in wheat through the mutation of a homeobox gene.
Human and Animal Cells EGR-G101(AES0182) Seminal vesicle secretory protein 7, PATE4, is not required for sperm function but for copulatory plug formation to ensure fecundity†.
Drosophila Drosophila melanogaster tPlus3a and tPlus3b ensure full male fertility by regulating transcription of Y-chromosomal, seminal fluid, and heat shock genes.
Drosophila New genes often acquire male-specific functions but rarely become essential in Drosophila.
Drosophila Identification of seven genes essential for male fertility through a genome-wide association study of non-obstructive azoospermia and RNA interference-mediated large-scale functional screening in Drosophila.
Drosophila DGRC#115192 , DGRC#122107 The PTK7-related transmembrane proteins off-track and off-track 2 are co-receptors for Drosophila Wnt2 required for male fertility.
Drosophila The Drosophila homologue of tubulin-specific chaperone E-like protein is required for synchronous sperm individualization and normal male fertility.
Mice RBRC00494 Polygenic expression of teratozoospermia and normal fertility in B10.MOL-TEN1 mouse strain.
Drosophila Defective transfer of seminal-fluid materials during matings of semi-fertile fruitless mutants in Drosophila.
DNA material pCALNL5 (RDB01862) Oocyte-specific overexpression of mouse bone morphogenetic protein-15 leads to accelerated folliculogenesis and an early onset of acyclicity in transgenic mice.