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  • 検索条件 : 絞込み (MeSH = Histone-Lysine N-Methyltransferase / genetics*)
生物種 リソース名 タイトル
線虫 tm1458 DOT-1.1-dependent H3K79 methylation promotes normal meiotic progression and meiotic checkpoint function in C. elegans.
研究用ヒト臍帯血幹細胞 Gene rearrangements of MLL and RUNX1 sporadically occur in normal CD34+ cells under cytokine stimulation.
ヒト・動物細胞 JHOS-2(RCB1521) , JHOS-3(RCB1546) , JHOS-4(RCB1678) Epigenetic Modifier SETD8 as a Therapeutic Target for High-Grade Serous Ovarian Cancer.
ショウジョウバエ Overexpression of dJmj differentially affects intestinal stem cells and differentiated enterocytes.
酵母 FY6986 , FY7014 , FY7132 , FY7273 , FY7520 , FYP466 Set7 Is a H3K37 Methyltransferase in Schizosaccharomyces pombe and Is Required for Proper Gametogenesis.
ヒト・動物細胞 TE-4(RCB2097) Small molecule inhibitors and CRISPR/Cas9 mutagenesis demonstrate that SMYD2 and SMYD3 activity are dispensable for autonomous cancer cell proliferation.
線虫 tm1821 Developmental Dynamics of X-Chromosome Dosage Compensation by the DCC and H4K20me1 in C. elegans.
線虫 tm1630 , tm3463 A role for Set1/MLL-related components in epigenetic regulation of the Caenorhabditis elegans germ line.
線虫 tm1821 H4K20me1 contributes to downregulation of X-linked genes for C. elegans dosage compensation.
線虫 tm1458 Histone methyltransferases MES-4 and MET-1 promote meiotic checkpoint activation in Caenorhabditis elegans.
線虫 tm1276 , tm1458 , tm1813 , tm574 Caenorhabditis elegans histone methyltransferase MET-2 shields the male X chromosome from checkpoint machinery and mediates meiotic sex chromosome inactivation.
ショウジョウバエ Genomewide identification of target genes of histone methyltransferase dG9a during Drosophila embryogenesis.
実験動物マウス RBRC00208 Prdm9 polymorphism unveils mouse evolutionary tracks.
ショウジョウバエ The Drosophila ortholog of MLL3 and MLL4, trithorax related, functions as a negative regulator of tissue growth.
ショウジョウバエ Histone recognition and nuclear receptor co-activator functions of Drosophila cara mitad, a homolog of the N-terminal portion of mammalian MLL2 and MLL3.