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  • 13 Hits
  • Search Condition : Filter (Species = Mice AND Author's Country of Origin = Australia)
Species Resource
Mice IRF-9 Knockout mouse , C57BL/6J The type I interferon-alpha mediates a more severe neurological disease in the absence of the canonical signaling molecule interferon regulatory factor 9.
Mice GFP-LC3#53(RBRC00806) Differential use of autophagy by primary dendritic cells specialized in cross-presentation.
Mice TP1-Venus (RBRC06137) Barx2 and Pax7 Regulate Axin2 Expression in Myoblasts by Interaction with β-Catenin and Chromatin Remodelling.
Mice Arx Dp-7 KI (B6)(RBRC03653) , Arx (GCG)7-1 KI (B6)(RBRC03654) Reduced polyalanine-expanded Arx mutant protein in developing mouse subpallium alters Lmo1 transcriptional regulation.
Mice C57BL/6-Pdcd1-/- mouse(RBRC02142) PD-1 dependent exhaustion of CD8+ T cells drives chronic malaria.
Mice delta-neo-Ripply2-KO(RBRC02227) Compound heterozygous mutations in RIPPLY2 associated with vertebral segmentation defects.
Mice B6.Cg-Tg(FucciS/G2/M)#492Bsi(RBRC02705) , B6.Cg-Tg(FucciG1)#639Bsi(RBRC02709) Stretched cell cycle model for proliferating lymphocytes.
Mice IRF-7 knockout mice , C57BL/6J (RBRC01420) Interferon regulatory factor 7 (IRF7) is required for the optimal initial control but not subsequent clearance of lymphocytic choriomeningitis virus infection in mice.
Mice semaphorin III/D (lacZ) Targeting mice (RBRC01105) The guidance molecule Semaphorin3A is differentially involved in the arealization of the mouse and primate neocortex.
Mice IRF-9 knockout mouse , C57BL/6J Mice deficient in STAT1 but not STAT2 or IRF9 develop a lethal CD4+ T-cell-mediated disease following infection with lymphocytic choriomeningitis virus.
Mice IRF-9 Knockout mouse , C57BL/6J(RBRC00916) , IRF-7 knockout mice , C57BL/6J(RBRC01420) IRF7-dependent type I interferon production induces lethal immune-mediated disease in STAT1 knockout mice infected with lymphocytic choriomeningitis virus.
Mice B6.Cg-Tg(FucciS/G2/M)#492Bsi(RBRC02705) , B6.Cg-Tg(FucciG1)#639Bsi(RBRC02709) T cell signaling. Antigen affinity, costimulation, and cytokine inputs sum linearly to amplify T cell expansion.
Mice TP1-venus (ICR)(RBRC06137) β-catenin is essential for differentiation of primary myoblasts via cooperation with MyoD and α-catenin.