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  • Search Condition : Filter (Species = Mice AND Resource = B6.Cg-Tg(CAG-Cre)CZ-MO2Osb(RBRC01828))
Species Resource
Mice B6.Cg-Tg(CAG-Cre)CZ-MO2Osb(RBRC01828) Disruption of the Sjögren-Larsson Syndrome Gene Aldh3a2 in Mice Increases Keratinocyte Growth and Retards Skin Barrier Recovery.
Mice B6.Cg-Tg(CAG-floxed Neo-EGFP)REP08Osb(RBRC02096) , B6-Tg(CAG-FLPe)36(RBRC01834) , B6.Cg-Tg(CAG-Cre)CZ-MO2Osb(RBRC01828) Hyaluronan deficiency in tumor stroma impairs macrophage trafficking and tumor neovascularization.
Mice B6.Cg-Tg(CAG-Cre)CZ-MO2Osb(RBRC01828) New BRAF knockin mice provide a pathogenetic mechanism of developmental defects and a therapeutic approach in cardio-facio-cutaneous syndrome.
Mice B6.Cg-Tg(CAG-Cre)CZ-MO2Osb(RBRC01828) Single-cell imaging of caspase-1 dynamics reveals an all-or-none inflammasome signaling response.
Mice B6.Cg-Tg(CAG-Cre)CZ-MO2Osb(RBRC01828) , C57BL/6-Tg(CAG-flpe)36Ito/ItoRbrc(RBRC01834) Laminin γ1 C-terminal Glu to Gln mutation induces early postimplantation lethality.
Mice B6.Cg-Tg(CAG-Cre)CZ-MO2Osb(RBRC01828) Loss of DDHD2, whose mutation causes spastic paraplegia, promotes reactive oxygen species generation and apoptosis.
Mice B6.Cg-Tg(CAG-Cre)CZ-MO2Osb(RBRC01828) Impaired epidermal permeability barrier in mice lacking elovl1, the gene responsible for very-long-chain fatty acid production.
Mice B6.Cg-Tg(CAG-Cre)CZ-MO2Osb(RBRC01828) Mice with an Oncogenic HRAS Mutation are Resistant to High-Fat Diet-Induced Obesity and Exhibit Impaired Hepatic Energy Homeostasis.
Mice B6.Cg-Tg(CAG-Cre)CZ-MO2Osb(RBRC01828) CLICK: one-step generation of conditional knockout mice.
Mice B6.Cg-Tg(CAG-Cre)CZ-MO2Osb(RBRC01828) Propagation of trimethylated H3K27 regulated by polycomb protein EED is required for embryogenesis, hematopoietic maintenance, and tumor suppression.
Mice B6.Cg-Tg(CAG-Cre)CZ-MO2Osb(RBRC01828) The protein phosphatase 6 catalytic subunit (Ppp6c) is indispensable for proper post-implantation embryogenesis.
Mice B6.Cg-Tg(CAG-Cre)CZ-MO2Osb(RBRC01828) ALK(R1275Q) perturbs extracellular matrix, enhances cell invasion and leads to the development of neuroblastoma in cooperation with MYCN.