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  • 検索条件 : 絞込み (生物種 = 実験動物マウス AND リソース名 = GFP-LC3#53 (RBRC00806))
生物種 リソース名
実験動物マウス GFP-LC3#53 (RBRC00806) Participation of autophagy in storage of lysosomes in neurons from mouse models of neuronal ceroid-lipofuscinoses (Batten disease).
実験動物マウス GFP-LC3#53 (RBRC00806) Autophagy regulates inflammation in adipocytes.
実験動物マウス GFP-LC3#53 (RBRC00806) Proteasome activity and autophagosome content in liver are reciprocally regulated by ethanol treatment.
実験動物マウス GFP-LC3#53 (RBRC00806) Autophagy limits acute myocardial infarction induced by permanent coronary artery occlusion.
実験動物マウス GFP-LC3#53 (RBRC00806) Autophagosomes initiate distally and mature during transport toward the cell soma in primary neurons.
実験動物マウス ERAI マウス (RBRC01099) , GFP-LC3#53 (RBRC00806) High resolution intravital imaging of subcellular structures of mouse abdominal organs using a microstage device.
実験動物マウス GFP-LC3#53 (RBRC00806) Autophagy in the intestinal epithelium regulates Citrobacter rodentium infection.
実験動物マウス GFP-LC3#53 (RBRC00806) Autophagy in the intestinal epithelium is not involved in the pathogenesis of intestinal tumors.
実験動物マウス GFP-LC3#53 (RBRC00806) Lc3 over-expression improves survival and attenuates lung injury through increasing autophagosomal clearance in septic mice.
実験動物マウス GFP-LC3#53 (RBRC00806) Prior starvation mitigates acute doxorubicin cardiotoxicity through restoration of autophagy in affected cardiomyocytes.
実験動物マウス GFP-LC3#53 (RBRC00806) Cathepsin-L, a key molecule in the pathogenesis of drug-induced and I-cell disease-mediated gingival overgrowth: a study with cathepsin-L-deficient mice.
実験動物マウス GFP-LC3#53 (RBRC00806) In vivo analysis of autophagy in response to nutrient starvation using transgenic mice expressing a fluorescent autophagosome marker.
実験動物マウス GFP-LC3#53 (RBRC00806) Immunohistochemical analysis of macroautophagy: recommendations and limitations.
実験動物マウス GFP-LC3#53 (RBRC00806) Phospholipase C-related catalytically inactive protein, a novel microtubule-associated protein 1 light chain 3-binding protein, negatively regulates autophagosome formation.