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  • 検索条件 : 絞込み (生物種 = 実験動物マウス AND リソース名 = IRF-3 Knockout mice)
生物種 リソース名
実験動物マウス IRF-3 Knockout mice , C57BL/6J Interferon response factor 3 is essential for house dust mite-induced airway allergy.
実験動物マウス IRF-3 Knockout mice , C57BL/6J Double-stranded RNA induces pancreatic beta-cell apoptosis by activation of the toll-like receptor 3 and interferon regulatory factor 3 pathways.
実験動物マウス IRF-3 Knockout mice , C57BL/6J Stimulator of IFN gene is critical for induction of IFN-beta during Chlamydia muridarum infection.
実験動物マウス IRF-3 Knockout mice , C57BL/6J Critical role of the IFN-stimulated gene factor 3 complex in TLR-mediated IL-27p28 gene expression revealing a two-step activation process.
実験動物マウス IRF-3 Knockout mice , C57BL/6J Baculovirus induces type I interferon production through toll-like receptor-dependent and -independent pathways in a cell-type-specific manner.
実験動物マウス IRF-3 Knockout mice , C57BL/6J Systemic lipopolysaccharide protects the brain from ischemic injury by reprogramming the response of the brain to stroke: a critical role for IRF3.
実験動物マウス IRF-3 Knockout mice , C57BL/6J The role of TBK1 and IKKε in the expression and activation of Pellino 1.
実験動物マウス IRF-3 Knockout mice , C57BL/6J (RBRC00858) Interferon regulatory factor 3 deficiency leads to interleukin-17-mediated liver ischemia-reperfusion injury.
実験動物マウス IRF-3 Knockout mice , C57BL/6J Paramyxovirus Sendai virus V protein counteracts innate virus clearance through IRF-3 activation, but not via interferon, in mice.
実験動物マウス IRF-3 Knockout mice , C57BL/6J Studies on the paramyxovirus accessory genes by reverse genetics in the Sendai virus-mouse system.
実験動物マウス IRF-3 Knockout mice , C57BL/6J(RBRC00858) Interferon regulatory factor 3 controls interleukin-17 expression in CD8 T lymphocytes.
実験動物マウス IRF-3 Knockout mice Interferon regulatory factor 3 is involved in Toll-like receptor 4 (TLR4)- and TLR3-induced IL-12p35 gene activation.
実験動物マウス IRF-3 Knockout mice , C57BL/6J(RBRC00858) Interferon regulatory factor 3 protects against adverse neo-intima formation.