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  • 検索条件 : 絞込み (生物種 = 遺伝子材料 AND 著者の国 = Italy)
生物種 リソース名
遺伝子材料 pCMV-SP1 (RDB04267) , pGEX-SP1 (RDB05699) , pGEX-hMAZ (RDB05701) , pCMV-MAZ (RDB03219). G4-DNA formation in the HRAS promoter and rational design of decoy oligonucleotides for cancer therapy.
遺伝子材料 hTRAIL/pMKITneo human TRAIL cDNA (RDB02934) Human CD34+ cells engineered to express membrane-bound tumor necrosis factor-related apoptosis-inducing ligand target both tumor cells and tumor vasculature.
遺伝子材料 pGEX-hMAZ (RDB03219) , pCMV-MAZ (RDB05701). The KRAS promoter responds to Myc-associated zinc finger and poly(ADP-ribose) polymerase 1 proteins, which recognize a critical quadruplex-forming GA-element.
遺伝子材料 pBluescript SKII+mIL10 (RDB01476) Viral and murine interleukin-10 are correctly processed and retain their biological activity when produced in tobacco.
遺伝子材料 pBluescript SKII+mIL10 (RDB01476) Development of an optimized tetracycline-inducible expression system to increase the accumulation of interleukin-10 in tobacco BY-2 suspension cells.
遺伝子材料 hTRAIL/pMKITneo (RDB02934) Surveillance of spontaneous breast cancer metastasis by TRAIL-expressing CD34⁺ cells in a xenograft model.
遺伝子材料 pBMSA-NP (RDB01972) Heat-shock protein 70 from plant biofactories of recombinant antigens activate multiepitope-targeted immune responses.
遺伝子材料 pCMV-MAZ (RDB03219) , pGEX-hMAZ (RDB05701) MAZ-binding G4-decoy with locked nucleic acid and twisted intercalating nucleic acid modifications suppresses KRAS in pancreatic cancer cells and delays tumor growth in mice.
遺伝子材料 pGL4-phPLAU (RDB07487) , pGL4-phSERPINE1(PAI-1) (RDB07461). HMGA1 regulates the Plasminogen activation system in the secretome of breast cancer cells.
遺伝子材料 pGL4-phIL6 (RDB07313) Chromatin remodelling and autocrine TNFα are required for optimal interleukin-6 expression in activated human neutrophils.
遺伝子材料 pbovLIF (RDB01660) Ultrastructural and histochemical features of the ground substance in cyclosporin A-induced gingival overgrowth.
遺伝子材料 hTRAIL/pMKITneo (RDB02934) YM155 sensitizes triple-negative breast cancer to membrane-bound TRAIL through p38 MAPK- and CHOP-mediated DR5 upregulation.