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  • 検索条件 : 絞込み (生物種 = 病原細菌 AND 著者の国 = United States of America)
生物種 リソース名 タイトル
病原細菌 V. parahaemolyticus RIMD2210633 A gatekeeper protein contributes to T3SS2 function via interaction with an ATPase in Vibrio parahaemolyticus.
病原細菌 JNBP_30766 Regulatory Small RNA Qrr2 Is Expressed Independently of Sigma Factor-54 and Can Function as the Sole Qrr Small RNA To Control Quorum Sensing in Vibrio parahaemolyticus.
病原細菌 A distinct inhibitory mechanism of the V-ATPase by Vibrio VopQ revealed by cryo-EM.
病原細菌 RIMD 2210633 Vibrio deploys type 2 secreted lipase to esterify cholesterol with host fatty acids and mediate cell egress.
病原細菌 V. parahaemolyticus RMID 2210633 Nucleolar c-Myc recruitment by a Vibrio T3SS effector promotes host cell proliferation and bacterial virulence.
病原細菌 RIMD 2210633 S-nitrosylation-mediated activation of a histidine kinase represses the type 3 secretion system and promotes virulence of an enteric pathogen.
病原細菌 Manipulation of IRE1-Dependent MAPK Signaling by a Vibrio Agonist-Antagonist Effector Pair.
病原細菌 RIMD 2210633 Transcriptomic analysis of PhoR reveals its role in regulation of swarming motility and T3SS expression in Vibrio parahaemolyticus.
病原細菌 Vibrio parahaemolyticus RIMD2210633 Carbohydrate metabolic systems present on genomic islands are lost and gained in Vibrio parahaemolyticus.
病原細菌 Vibrio parahaemolyticus RIMD2210633 Quorum Sensing Regulators AphA and OpaR Control Expression of the Operon Responsible for Biosynthesis of the Compatible Solute Ectoine.
病原細菌 Unsupervised Learning Approach for Comparing Multiple Transposon Insertion Sequencing Studies.
病原細菌 CRISPR-Cas systems are present predominantly on mobile genetic elements in Vibrio species.
病原細菌 Vibrio parahaemolyticus Natural Transformation in Vibrio parahaemolyticus: a Rapid Method To Create Genetic Deletions.
病原細菌 病原細菌 Mechanisms for Pseudoalteromonas piscicida-Induced Killing of Vibrios and Other Bacterial Pathogens.
病原細菌 病原細菌 Acute Hepatopancreatic Necrosis Disease-Causing Vibrio parahaemolyticus Strains Maintain an Antibacterial Type VI Secretion System with Versatile Effector Repertoires.
病原細菌 病原細菌 The cytotoxic type 3 secretion system 1 of Vibrio rewires host gene expression to subvert cell death and activate cell survival pathways.
病原細菌 病原細菌 T3SS effector VopL inhibits the host ROS response, promoting the intracellular survival of Vibrio parahaemolyticus.