• 14 Hits
  • 検索条件 : 絞込み (MeSH = Pyrimidines / administration & dosage)
生物種 リソース名 タイトル
ヒト・動物細胞 WEHI-3(RCB0035) , Ba/F3(RCB0805) Synergistic killing effect of imatinib and simvastatin on imatinib-resistant chronic myelogenous leukemia cells.
ヒト・動物細胞 Ba/F3 Mechanisms of Resistance to NTRK Inhibitors and Therapeutic Strategies in NTRK1-Rearranged Cancers.
ヒト・動物細胞 Btk^(-) DT40(RCB1468) Selinexor is effective in acquired resistance to ibrutinib and synergizes with ibrutinib in chronic lymphocytic leukemia.
ヒト・動物細胞 KP4(RCB1005) Translational pharmacokinetic-pharmacodynamic modeling of preclinical and clinical data of the oral MET inhibitor tepotinib to determine the recommended phase II dose.
ヒト・動物細胞 CACO-2(RCB0988) Augmented pentose phosphate pathway plays critical roles in colorectal carcinomas.
ヒト・動物細胞 JHOC-5(RCB1520) Low-dose triple drug combination targeting the PI3K/AKT/mTOR pathway and the MAPK pathway is an effective approach in ovarian clear cell carcinoma.
ヒト・動物細胞 StromaNKtert(RCB2350) Bruton's tyrosine kinase inhibition increases BCL-2 dependence and enhances sensitivity to venetoclax in chronic lymphocytic leukemia.
ヒト・動物細胞 UV♀2(RCB1994) Dual inhibition of mTORC1 and mTORC2 perturbs cytoskeletal organization and impairs endothelial cell elongation.
ヒト・動物細胞 PK-59(RCB1901) Pharmacokinetic evaluation of the PNC disassembler metarrestin in wild-type and Pdx1-Cre;LSL-KrasG12D/+;Tp53R172H/+ (KPC) mice, a genetically engineered model of pancreatic cancer.
ヒト・動物細胞 MIA Paca2(RCB2094) , PANC-1(RCB2095) Enhancement of cytotoxic effects of gemcitabine by Dclk1 inhibition through suppression of Chk1 phosphorylation in human pancreatic cancer cells.
病原微生物 NA Scedosporium aurantiacum brain abscess after near-drowning in a survivor of a tsunami in Japan.
ヒト・動物細胞 LoVo(RCB1639) , HCT116(RCB2979) EMD 1214063 and EMD 1204831 constitute a new class of potent and highly selective c-Met inhibitors.
病原微生物 IFM 58108 [Chromoblastomycosis caused by Fonsecaea monophora].
ヒト・動物細胞 K562(RCB0027) Depsipeptide enhances imatinib mesylate-induced apoptosis of Bcr-Abl-positive cells and ectopic expression of cyclin D1, c-Myc or active MEK abrogates this effect.