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論文 - 一覧

  • 11 Hits
  • 検索条件 : 絞込み (MeSH = Inclusion Bodies / metabolism)
生物種 リソース名 タイトル
ヒト・動物細胞 , 遺伝子材料 201B7(HPS0063) , CSII-EF-MCS-IRES2-Venus (RDB04384) , pCMV-VSV-G-RSV-Rev (RDB04393) , pCAG-HIVgp (RDB04394) Monomerization of TDP-43 is a key determinant for inducing TDP-43 pathology in amyotrophic lateral sclerosis.
ヒト・動物細胞 A-THP-1(RCB2128) A53T mutant α-synuclein fibrils formed in macrophage are spread to neurons.
ヒト・動物細胞 KG-1-C(RCB0270) Suppression of dynamin GTPase decreases α-synuclein uptake by neuronal and oligodendroglial cells: a potent therapeutic target for synucleinopathy.
遺伝子材料 AxDsR-WT.TDP43 (RDB15499) , AxDsR-CTF.TDP43 (RDB15500) Adenoviral expression of TDP-43 and FUS genes and shRNAs for protein degradation pathways in rodent motoneurons in vitro and in vivo.
一般微生物 JCM 10015 Characterization and identification of the proteins bound to two types of polyhydroxyalkanoate granules in Pseudomonas sp. 61-3.
遺伝子材料 GFP-OPTN-WT (RDB16707) , GFP-OPTN-E50K (RDB16708) , GFP-OPTN-E478G (RDB16709) Determination of cytoplasmic optineurin foci sizes using image correlation spectroscopy.
遺伝子材料 CMV:TDP43-GFP (RDB15742) , CMV:GFP-TDP25 (RDB15743) , CMV:GFP-NLS-TDP25 (RDB15744) , CMV:GFP-NLSNP-TDP25 (RDB15745) , GFP-NLS NP (RDB15746) , mCherry-Ubiquitin (RDB15747). Interaction of RNA with a C-terminal fragment of the amyotrophic lateral sclerosis-associated TDP43 reduces cytotoxicity.
ヒト・動物細胞 HuH-7(RCB1366) Autophagy activation by rapamycin eliminates mouse Mallory-Denk bodies and blocks their proteasome inhibitor-mediated formation.
ヒト・動物細胞 CACO-2(RCB0988) Efficient solubilization, activation, and purification of recombinant Cry45Aa of Bacillus thuringiensis expressed as inclusion bodies in Escherichia coli.
遺伝子材料 pSHY13-2 (RDB08867) , pSHY15-1 (RDB08869) , pSHY14-5 (RDB08868) Inhibition of endoplasmic reticulum (ER)-to-Golgi transport induces relocalization of binding protein (BiP) within the ER to form the BiP bodies.
ショウジョウバエ Protein transduction domain-mediated delivery of QBP1 suppresses polyglutamine-induced neurodegeneration in vivo.