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  • 検索条件 : 絞込み (生物種 = 遺伝子材料 AND リソース名 = CSII-CMV-MCS-IRES2-Bsd(RDB04385))
生物種 リソース名 タイトル
遺伝子材料 pCAGGS (RDB08938) , pCMV-VSV-G-RSV-Rev (RDB04393) , pCAG-HIVgp (RDB04394) , CSII-CMV-MCS-IRES2-Bsd (RDB04385) An interactome-based framework for DDB1- and CUL4-associated factor prioritization in targeted protein degradation.
遺伝子材料 CSII-CMV-MCS-IRES2-Bsd (RDB04385) , pCAG-HIVgp (RDB04394) , pCMV-VSV-G-RSV-Rev (RDB04393) ALLO-1a is a ubiquitin-binding adaptor for allophagy in Caenorhabditis elegans.
遺伝子材料 CSII-CMV-MCS-IRES2-Bsd (RDB04385) Innovative Methods to Label, Track, and Quantitate Extracellular Vesicle Uptake.
遺伝子材料 CSII-CMV-MCS-IRES2-Bsd (RDB04385) , pCMV-VSV-G-RSV-Rev (RDB04393) , pCAG-HIVgp (RDB04394) TRBP modulates RLR signaling by inhibiting PKR-mediated antiviral stress granule formation.
ヒト・動物細胞 , 遺伝子材料 293T(RCB2202) , CSII-CMV-MCS-IRES2-Bsd (RDB04385) SARS-CoV-2 inhibition through mRNA delivery using engineered extracellular vesicles displaying the spike protein.
ヒト・動物細胞 , 遺伝子材料 293T(RCB2202) , CSII-CMV-MCS-IRES2-Bsd (RDB04385) Synergistic antiviral activity of a cathepsin B/L inhibitor and a TMPRSS2 inhibitor against SARS-CoV-2 in vitro and in vivo.
遺伝子材料 CSII-CMV-MCS-IRES2-Bsd (RDB04385) , pCMV-VSV-G-RSV-Rev (RDB04393) , pCAG-HIVgp (RDB04394) Atorvastatin exhibits anticancer effects by inhibiting YAP/TAZ activity in mesenchymal-like non-small cell lung cancer.
遺伝子材料 CSII-CMV-MCS-IRES2-Bsd (RDB04385) , pCMV-VSV-G-RSV-Rev (RDB04393) , pCAG-HIVgp (RDB04394) Biomechanical control of vascular morphogenesis by the surrounding stiffness.
遺伝子材料 , ヒト・動物細胞 CSII-CMV-MCS-IRES2-Bsd (RDB04385) , pCMV-VSV-G-RSV-Rev (RDB04393) , pCAG-HIVgp (RDB04394) , JMSU1(RCB2227) AEBP1-GLI1 pathway attenuates the FACT complex dependency of bladder cancer cell survival.
ヒト・動物細胞 , 遺伝子材料 CTLL-2(RCB0637) , 293T(RCB2202) , CSII-CMV-MCS-IRES2-Bsd (RDB04385) , pCMV-VSV-G-RSV-Rev (RDB04393) , pCAG-HIVgp (RDB04394) Expression and localization of lysosomal-associated membrane protein 1- and perforin-based hybrid molecules in the murine cytotoxic T cell line CTLL-2.
遺伝子材料 , ヒト・動物細胞 CSII-CMV-MCS-IRES2-Bsd (RDB04385) , pCMV-VSV-G-RSV-Rev (RDB04393) , pCAG-HIVgp (RDB04394) , 201B7(HPS0063) Sustained chromosomal passenger complex activity preserves the pluripotency of human embryonic carcinoma cells.
遺伝子材料 CSII-CMV-AFF4T254S-IRES2-Bsd (RDB21100) , pCAG-HIVgp (RDB04394) , pCMV-VSV-G-RSV-Rev (RDB04393) , CSII-CMV-MCS-IRES2-Bsd (RDB04385) A common molecular mechanism underlying Cornelia de Lange and CHOPS syndromes.
遺伝子材料 CSII-CMV-MCS-IRES2-Bsd (RDB04385) , pCAG-HIVgp (RDB04394) , pCMV-VSV-G-RSV-Rev (RDB04393) PARP inhibition-associated heterochromatin confers increased DNA replication stress and vulnerability to ATR inhibition in SMARCA4-deficient cells.
遺伝子材料 CSII-CMV-MCS-IRES2-Bsd (RDB04385) RNF168 E3 ligase participates in ubiquitin signaling and recruitment of SLX4 during DNA crosslink repair.
ヒト・動物細胞 , 遺伝子材料 Hep G2 , NEC8(RCB0489) , 293T(RCB2202) , CHO-K1(RCB0285) , S2 (Drosophila)(RCB1153) , CSII-CMV-MCS-IRES2-Bsd (RDB04385) Identification of C-mannosylation in a receptor tyrosine kinase AXL.
遺伝子材料 CSII-CMV-MCS-IRES2-Bsd (RDB04385) Polyubiquitinated PCNA triggers SLX4-mediated break-induced replication in alternative lengthening of telomeres (ALT) cancer cells.
遺伝子材料 CSII-CMV-MCS-IRES2-Bsd (RDB04385) SMARCA4 deficiency-associated heterochromatin induces intrinsic DNA replication stress and susceptibility to ATR inhibition in lung adenocarcinoma.
遺伝子材料 CSII-CMV-MCS-IRES2-Bsd (RDB04385) , pCMV-VSV-G-RSV-Rev (RDB04393) , pCAG-HIVgp (RDB04394) Susceptibility of rat immortalized neuronal cell line Rn33B expressing equine major histocompatibility class 1 to equine herpesvirus-1 infection is differentiation dependent.
遺伝子材料 CSII-CMV-MCS-IRES2-Bsd (RDB04385) , pCMV-VSV-G-RSV-Rev (RDB04393) , pCAG-HIVgp (RDB04394) Development of Human CBF1-Targeting Single-Stranded DNA Aptamers with Antiangiogenic Activity In Vitro.
遺伝子材料 CSII-CMV-MCS-IRES2-Bsd (RDB04385) , pCMV-VSV-G-RSV-Rev (RDB04393) , pCAG-HIVgp (RDB04394) PSMA-positive membranes secreted from prostate cancer cells have potency to transform vascular endothelial cells into an angiogenic state.