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Species Resource Title
C.elegans Feeding state-dependent regulation of developmental plasticity via CaMKI and neuroendocrine signaling.
C.elegans tm2308 , tm2482 , tm629 The unfolded protein response in a pair of sensory neurons promotes entry of C. elegans into dauer diapause.
C.elegans tm2654 , tm1944 Loss of a neural AMP-activated kinase mimics the effects of elevated serotonin on fat, movement, and hormonal secretions.
C.elegans tm2308 , tm3620 , tm2560 , tm2416 , tm2001 , tm3618 , tm1983 , tm2988 Ins-4 and daf-28 function redundantly to regulate C. elegans L1 arrest.
C.elegans tm1907 , tm2308 , tm2416 Diapause is associated with a change in the polarity of secretion of insulin-like peptides.
C.elegans tm4574 Mitochondrial function is required for secretion of DAF-28/insulin in C. elegans.
C.elegans tm2416 , tm2308 Specific insulin-like peptides encode sensory information to regulate distinct developmental processes.
C.elegans tm2308 The thioredoxin TRX-1 modulates the function of the insulin-like neuropeptide DAF-28 during dauer formation in Caenorhabditis elegans.
C.elegans tm2308 , tm1888 ASNA-1 positively regulates insulin secretion in C. elegans and mammalian cells.