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  • 14 Hits
  • 検索条件 : 絞込み (MeSH = Matrix Metalloproteinases / genetics)
生物種 リソース名 タイトル
ヒト・動物細胞 Hep G2 Hypoxia accelerates cancer invasion of hepatoma cells by upregulating MMP expression in an HIF-1alpha-independent manner.
カタユウレイボヤ・(ニッポンウミシダ) Wild C. int Cionin, a vertebrate cholecystokinin/gastrin homolog, induces ovulation in the ascidian Ciona intestinalis type A.
ヒト・動物細胞 LM8(RCB1450) Reduction of metastasis, cell invasion, and adhesion in mouse osteosarcoma by YM529/ONO-5920-induced blockade of the Ras/MEK/ERK and Ras/PI3K/Akt pathway.
ヒト・動物細胞 PMF-ko14(RCB1426) The Promoting Effect of the Extracellular Matrix Peptide TNIIIA2 Derived from Tenascin-C in Colon Cancer Cell Infiltration.
ヒト・動物細胞 HCE-T(RCB2280) Expression, Regulation, and Effects of Interleukin-17f in the Human Ocular Surface.
メダカ Hatching enzyme Developmental mechanisms of migratory muscle precursors in medaka pectoral fin formation.
ツメガエル・イモリ Upregulation of matrix metalloproteinase triggers transdifferentiation of retinal pigmented epithelial cells in Xenopus laevis: A Link between inflammatory response and regeneration.
ツメガエル・イモリ A unique role of thyroid hormone receptor β in regulating notochord resorption during Xenopus metamorphosis.
ヒト・動物細胞 WERI-Rb-1(RCB2146) , Y79(RCB1645) Molecular deregulation induced by silencing of the high mobility group protein A2 gene in retinoblastoma cells.
ヒト・動物細胞 MRC-5 Tumor-stromal cell interaction under hypoxia increases the invasiveness of pancreatic cancer cells through the hepatocyte growth factor/c-Met pathway.
ヒト・動物細胞 MH7A(RCB1512) Model-based analysis of matrix metalloproteinase expression under mechanical shear.
メダカ OK-Cab (MT830) , Database TGFβ-2 signaling is essential for osteoblast migration and differentiation during fracture healing in medaka fish.
ヒト・動物細胞 RAW 264(RCB0535) Astaxanthin suppresses scavenger receptor expression and matrix metalloproteinase activity in macrophages.
ヒト・動物細胞 TKD2(RCB0752) , Hep G2 , A549(RCB0098) , Kato III(RCB2088) Induction of matrix metalloproteinase gene expression in an endothelial cell line by direct interaction with malignant cells.