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  • 検索条件 : 絞込み (MeSH = DNA End-Joining Repair*)
生物種 リソース名 タイトル
線虫 tm1524 Recruitment of MRE-11 to complex DNA damage is modulated by meiosis-specific chromosome organization.
線虫 tm1298 Disabling the Fanconi Anemia Pathway in Stem Cells Leads to Radioresistance and Genomic Instability.
ヒト・動物細胞 MC3T3-G2/PA6(RCB1127) Alternative NHEJ pathway proteins as components of MYCN oncogenic activity in human neural crest stem cell differentiation: implications for neuroblastoma initiation.
線虫 tm2026 BRCA1-associated structural variations are a consequence of polymerase theta-mediated end-joining.
線虫 tm1203 NHJ-1 Is Required for Canonical Nonhomologous End Joining in Caenorhabditis elegans.
線虫 tm2026 Translesion synthesis polymerases are dispensable for C. elegans reproduction but suppress genome scarring by polymerase theta-mediated end joining.
ヒト・動物細胞 A549(RCB0098) RNF8 promotes high linear energy transfer carbon-ion-induced DNA double-stranded break repair in serum-starved human cells.
ヒト・動物細胞 KGN(RCB1154) FOXL2 directs DNA double-strand break repair pathways by differentially interacting with Ku.
ヒト・動物細胞 HCT116(RCB2979) , CHO-K1 Feline XLF accumulates at DNA damage sites in a Ku-dependent manner.
遺伝子材料 , ヒト・動物細胞 p3XFLAG-CDCA7 WT (RDB17374) , p3XFLAG-CDCA7 R274C (RDB17375) , p3XFLAG-HELLS WT (RDB17376) , p3XFLAG-HELLS Q699R (RDB17377) , DNMT3B px330 (RDB17378) , ZBTB24 px330 (RDB17379) , CDCA7 px330 (RDB17380) , HELLS px330 (RDB17381) , Ku80 px330 (RDB17382) , HEV0190 CDCA7 and HELLS mutations undermine nonhomologous end joining in centromeric instability syndrome.
ショウジョウバエ Drosophila DNA polymerase theta utilizes both helicase-like and polymerase domains during microhomology-mediated end joining and interstrand crosslink repair.
線虫 tm1842 , tm1524 Interdependent and separable functions of Caenorhabditis elegans MRN-C complex members couple formation and repair of meiotic DSBs.
線虫 tm2026 Polymerase theta-mediated end joining of replication-associated DNA breaks in C. elegans.
線虫 tm2073 , tm1145 , tm1842 , tm2026 Polymerase Θ is a key driver of genome evolution and of CRISPR/Cas9-mediated mutagenesis.
カイコ CRISPR/Cas9-mediated knockout of factors in non-homologous end joining pathway enhances gene targeting in silkworm cells.
線虫 tm1842 Impaired resection of meiotic double-strand breaks channels repair to nonhomologous end joining in Caenorhabditis elegans.