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  • 12 Hits
  • 検索条件 : 絞込み (MeSH = Tandem Repeat Sequences)
生物種 リソース名 タイトル
病原真核微生物 Aspergillus fumigatus IFM62918, IFM63432, IFM64460, IFM64733 Development and validation of LAMP primer sets for rapid identification of Aspergillus fumigatus carrying the cyp51A TR46 azole resistance gene.
ヒト・動物細胞 32Dcl3(RCB1377) Co-expression of wild-type FLT3 attenuates the inhibitory effect of FLT3 inhibitor on FLT3 mutated leukemia cells.
病原真核微生物 IFM 63432, 64732 (= OKH50) Detection of Substitutions at 98th, 121st, and 289th Amino Acid Residues in Cyp51A using Cycling Probes.
ヒト・動物細胞 Ba/F3(RCB0805) , 32D(RCB1145) Mechanism of constitutive activation of FLT3 with internal tandem duplication in the juxtamembrane domain.
病原真核微生物 病原真菌：IFM 62916, IFM 63240, IFM 63241, IFM 63242, IFM 63243, IFM 63249, IFM 63537, IFM 63594, IFM 63595, IFM 63596, IFM 63714, IFM 64173, IFM 62140, IFM 64258, IFM 64303, IFM 63666, IFM 63768, IFM 63772 Current Status of Azole-resistant Aspergillus fumigatus Isolates in East Asia.
病原微生物 Sensitisation of an Azole-Resistant Aspergillus fumigatus Strain containing the Cyp51A-Related Mutation by Deleting the SrbA Gene.
遺伝子材料 pEF-T7-mouse Doc2 beta (RDB15101) , pEF-T7-mouse Doc2 gamma (RDB15102). The C2A domain of double C2 protein gamma contains a functional nuclear localization signal.
ヒト・動物細胞 32D(RCB1145) Selective apoptosis of tandemly duplicated FLT3-transformed leukemia cells by Hsp90 inhibitors.
ヒト・動物細胞 32D Activating mutation of D835 within the activation loop of FLT3 in human hematologic malignancies.
病原微生物 IFM 57372 , IFM 57373 , IFM 57374 , IFM 57376 , IFM 57377 , IFM 57378 , IFM 57379 , IFM 57380 , IFM 57381 , IFM 57382 , ... Genotyping of fluconazole-resistant Candida albicans isolated from Uighurian people in Xinjing (China) using ALTS/RFLP and micro-TGGE method.
イネ W1057 , W1577 , W1263 , W0008 Identification and diversity of functional centromere satellites in the wild rice species Oryza brachyantha.
シロイヌナズナ / 植物培養細胞・遺伝子 sjo02300 , sjo02100 , sjo02600 , sjs00500 , sjs00200 Molecular evolution of pentatricopeptide repeat genes reveals truncation in species lacking an editing target and structural domains under distinct selective pressures.