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  • Search Condition : Filter (MeSH = Brain / drug effects*)
Species Resource Title
Human and Animal Cells PC-12(RCB0009) Toxicity of internalized polyalanine to cells depends on aggregation.
Japanese macaques Deschloroclozapine, a potent and selective chemogenetic actuator enables rapid neuronal and behavioral modulations in mice and monkeys.
Human and Animal Cells MG6(RCB2403) Photo-oxygenation by a biocompatible catalyst reduces amyloid-β levels in Alzheimer's disease mice.
Human and Animal Cells NB2a(RCB2639) Trichloroethylene exposure aggravates behavioral abnormalities in mice that are deficient in superoxide dismutase.
Human and Animal Cells A431(RCB0202) Clozapine-dependent inhibition of EGF/neuregulin receptor (ErbB) kinases.
Human and Animal Cells LLC-GA5-CoL150(RCB0871) In vitro and in vivo evaluations of the P-glycoprotein-mediated efflux of dibenzoylhydrazines.
Mice RBRC00806 Prenatal alcohol exposure impairs autophagy in neonatal brain cortical microvessels.
Human and Animal Cells PC-12(RCB0009) Interaction of new antidepressants with sigma-1 receptor chaperones and their potentiation of neurite outgrowth in PC12 cells.
Human and Animal Cells Neuroprotective efficacy of the peroxisome proliferator-activated receptor delta-selective agonists in vitro and in vivo.
Silkworms Identification of 20-hydroxyecdysone-inducible genes from larval brain of the silkworm, Bombyx mori, and their expression analysis.
Human and Animal Cells PC-12(RCB0009) The neuroprotective effect of modified Yeoldahanso-tang via autophagy enhancement in models of Parkinson's disease.
Medaka Kyoto-cab (OK-Cab (MT830)) A chemical neurotoxin, MPTP induces Parkinson's disease like phenotype, movement disorders and persistent loss of dopamine neurons in medaka fish.
DNA material pRHO1 Rat heme oxygenase cDNA (RDB01208) Brain responses to acute withdrawal in phenobarbital-dependent rats.
Rats Pentosan polysulfate protects brain endothelial cells against bacterial lipopolysaccharide-induced damages.
Rats The effects of lithium chloride and other substances on levels of brain N-acetyl-L-aspartic acid in Canavan disease-like rats.