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論文 - 一覧

  • 14 Hits
  • 検索条件 : 絞込み (MeSH = Wound Healing / genetics)
生物種 リソース名 タイトル
ヒト・動物細胞 B16 , B16F10(RCB2630) The a3 isoform vacuolar type H⁺-ATPase promotes distant metastasis in the mouse B16 melanoma cells.
遺伝子材料 CS-RfA-ETBsd (RDB07917) , pENTR4-H1 (RDB04395) Neutrophil Extracellular Traps Delay Diabetic Wound Healing by Inducing Endothelial-to-Mesenchymal Transition via the Hippo pathway.
ヒト・動物細胞 293T(RCB2202) The role of the interaction of the vinculin proline-rich linker region with vinexin α in sensing the stiffness of the extracellular matrix.
ゼブラフィッシュ Tg(krt4:gal4; uas:gfp) Heterogeneous fates and dynamic rearrangement of regenerative epidermis-derived cells during zebrafish fin regeneration.
ヒト・動物細胞 HCE-T(RCB2280) NFE2L2 activator RS9 protects against corneal epithelial cell damage in dry eye models.
ショウジョウバエ DGRC#109007 Injury Activates a Dynamic Cytoprotective Network to Confer Stress Resilience and Drive Repair.
ショウジョウバエ 5680R-2 , DGRC#106948 , DGRC#106843 , DGRC#106646 , DGRC#106649 JNK-mediated Slit-Robo signaling facilitates epithelial wound repair by extruding dying cells.
ショウジョウバエ 12019R-2 cdc37 is essential for JNK pathway activation and wound closure in Drosophila.
ショウジョウバエ DGRC#111872 JNK-dependent cell cycle stalling in G2 promotes survival and senescence-like phenotypes in tissue stress.
ヒト・動物細胞 HL60(RCB0041) Identification of Specific miRNAs in Neutrophils of Type 2 Diabetic Mice: Overexpression of miRNA-129-2-3p Accelerates Diabetic Wound Healing.
ヒト・動物細胞 TE-5(RCB1949) , TE-8(RCB2098) , TE-10(RCB2099) , TE-15(RCB1951) Podoplanin is expressed at the invasive front of esophageal squamous cell carcinomas and is involved in collective cell invasion.
ヒト・動物細胞 AKT primes snail-induced EMT concomitantly with the collective migration of squamous cell carcinoma cells.
ショウジョウバエ 9553R-3 , 9553R-4 , 5680R-1 , 5680R-2 , 2275R-2 , 15509R-2 Transcriptional regulation of Profilin during wound closure in Drosophila larvae.
ヒト・動物細胞 C2C12(RCB0987) Notch2 negatively regulates myofibroblastic differentiation of myoblasts.