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論文 - 一覧

  • 14 Hits
  • 検索条件 : 絞込み (MeSH = rac1 GTP-Binding Protein / genetics)
生物種 リソース名 タイトル
ヒト・動物細胞 RAW 264(RCB0535) Development of an automated fluorescence microscopy system for photomanipulation of genetically encoded photoactivatable proteins (optogenetics) in live cells.
ヒト・動物細胞 RAW 264(RCB0535) Rab10-Positive Tubular Structures Represent a Novel Endocytic Pathway That Diverges From Canonical Macropinocytosis in RAW264 Macrophages.
ヒト・動物細胞 TE-1(RCB1894) , TE-8(RCB2098) Filamin C promotes lymphatic invasion and lymphatic metastasis and increases cell motility by regulating Rho GTPase in esophageal squamous cell carcinoma.
ヒト・動物細胞 HuH-7 Long noncoding RNA SchLAH suppresses metastasis of hepatocellular carcinoma through interacting with fused in sarcoma.
ヒト・動物細胞 HeLa(RCB0007) Optogenetic control of small GTPases reveals RhoA mediates intracellular calcium signaling.
線虫 tm1556 , tm5159 TIAM-1/GEF can shape somatosensory dendrites independently of its GEF activity by regulating F-actin localization.
ヒト・動物細胞 Suppression of Rac1 activity at the apical membrane of MDCK cells is essential for cyst structure maintenance.
ヒト・動物細胞 293(RCB1637) Modification of mineralocorticoid receptor function by Rac1 GTPase: implication in proteinuric kidney disease.
ヒト・動物細胞 Involvement of Rac and Rho signaling in cancer cell motility in 3D substrates.
ショウジョウバエ 14895R-1 , 2248R-2 , 9774R-2 , 18255R-3 Requirement for Pak3 in Rac1-induced organization of actin and myosin during Drosophila larval wound healing.
実験動物マウス RBRC02432 Angiotensin II- and salt-induced kidney injury through Rac1-mediated mineralocorticoid receptor activation.
遺伝子材料 AxCANLacZ (RDB1749) Over-expression of PAR-3 suppresses contact-mediated inhibition of cell migration in MDCK cells.
病原微生物 V. parahaemolyticus? Dominant-negative Rho, Rac, and Cdc42 facilitate the invasion process of Vibrio parahaemolyticus into Caco-2 cells.
病原微生物 S. deliver? Shigella deliver an effector protein to trigger host microtubule destabilization, which promotes Rac1 activity and efficient bacterial internalization.