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論文 - 一覧

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  • 検索条件 : 絞込み (MeSH = Rifampin / pharmacology)
生物種 リソース名 タイトル
ヒト・動物細胞 293T(RCB2202) NEAT1_2 and DAZAP1, Paraspeckle Components, Interact with PXR to Negatively Regulate CYP3A4 Induction.
原核生物(大腸菌) Suppression of the E. coli SOS response by dNTP pool changes.
ヒト・動物細胞 Threonine-290 regulates nuclear translocation of the human pregnane X receptor through its phosphorylation/dephosphorylation by Ca2+/calmodulin-dependent protein kinase II and protein phosphatase 1.
ヒト・動物細胞 Hep G2 Regulation of pregnane X receptor (PXR) function and UGT1A1 gene expression by posttranslational modification of PXR protein.
ヒト・動物細胞 CACO-2(RCB0988) Determination of p-glycoprotein ATPase activity using luciferase.
原核生物(大腸菌) pK18mobsacB Monooxygenation of rifampicin catalyzed by the rox gene product of Nocardia farcinica: structure elucidation, gene identification and role in drug resistance.
病原微生物 IFM 0150 , IFM 0236 , IFM 0239 , IFM 0290 , IFM 0319 , IFM 2057 Phosphorylative inactivation of rifampicin by Nocardia otitidiscaviarum.
原核生物(大腸菌) ME9385 DNA damage-inducible replication of the Escherichia coli chromosome is initiated at separable sites within the minimal oriC.
原核生物(大腸菌) ME9228 , ME9197 Mode of initiation of constitutive stable DNA replication in RNase H-defective mutants of Escherichia coli K-12.
原核生物(大腸菌) ME9340 Effects of oxygen stress on membrane functions in Escherichia coli: role of HPI catalase.
原核生物(大腸菌) ME6085 A new conditional lethal mutator (dnaQ49) in Escherichia coli K12.
ヒト・動物細胞 HOS(RCB0992) , Saos-2(RCB0428) Inhibitory effect of vitamin K(2) on interleukin-1beta-stimulated proliferation of human osteoblasts.
原核生物(大腸菌) ME9062 (BW25113) Inadequate inhibition of host RNA polymerase restricts T7 bacteriophage growth on hosts overexpressing udk.
原核生物(大腸菌) ME8079(AB1157) , ME8080(AB1885) Mutation spectrum induced by dihydropyrazines in Escherichia coli.
ヒト・動物細胞 LLC-GA5-CoL150(RCB0871) Functional induction and de-induction of P-glycoprotein by St. John's wort and its ingredients in a human colon adenocarcinoma cell line.