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  • Search Condition : Filter (MeSH = Muscle Cells / metabolism)
Species Resource
C.elegans tm752 Titin/connectin-related proteins in C. elegans: a review and new findings.
C.elegans tm1996 Functional characterization of three G protein-coupled receptors for pigment dispersing factors in Caenorhabditis elegans.
C.elegans tm1034 , tm1275 , tm469 Role of C. elegans TAT-1 protein in maintaining plasma membrane phosphatidylserine asymmetry.
Xenopus tropicalis Ivory Coast line Regulation of thyroid hormone sensitivity by differential expression of the thyroid hormone receptor during Xenopus metamorphosis.
Drosophila 31935R-1 Regulators of autophagosome formation in Drosophila muscles.
C.elegans tm6548 Neuronal reprograming of protein homeostasis by calcium-dependent regulation of the heat shock response.
C.elegans tm474 Functional phenotypic rescue of Caenorhabditis elegans neuroligin-deficient mutants by the human and rat NLGN1 genes.
C.elegans tm299 Cell architecture: surrounding muscle cells shape gland cell morphology in the Caenorhabditis elegans pharynx.
C.elegans tm2549 , tm1810 , tm356 , tm1083 , tm3569 , tm481 , tm2569 , tm3671 , tm861 A regulatory module controlling pharyngeal development and function in Caenorhabditis elegans.
C.elegans tm474 Neuroligin-deficient mutants of C. elegans have sensory processing deficits and are hypersensitive to oxidative stress and mercury toxicity.
DNA material AxCAmTSC-22 (forward) (RDB02760) , AxCALacZ (RDB01745) TSC-22 up-regulates collagen 3a1 gene expression in the rat heart.
Drosophila Stocks of DGRC Kytoto Stress and muscular dystrophy: a genetic screen for dystroglycan and dystrophin interactors in Drosophila identifies cellular stress response components.
Human and Animal Cells Vitamin D receptor gene silencing effects on differentiation of myogenic cell lines.
Human and Animal Cells 10T1/2 (RCB0247) CD271/p75(NTR) inhibits the differentiation of mesenchymal stem cells into osteogenic, adipogenic, chondrogenic, and myogenic lineages.
Human and Animal Cells GHRP-2, a GHS-R agonist, directly acts on myocytes to attenuate the dexamethasone-induced expressions of muscle-specific ubiquitin ligases, Atrogin-1 and MuRF1.