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  • 14 Hits
  • 検索条件 : 絞込み (MeSH = Arthritis, Rheumatoid / drug therapy*)
生物種 リソース名 タイトル
ヒト・動物細胞 MH7A(RCB1512) DPP4 reduces proinflammatory cytokine production in human rheumatoid arthritis synovial fibroblasts.
ヒト・動物細胞 MH7A(RCB1512) Effects of the novel immunosuppressant FTY720 in a murine rheumatoid arthritis model.
ヒト・動物細胞 MH7A(RCB1512) Antcin K Inhibits TNF-α, IL-1β and IL-8 Expression in Synovial Fibroblasts and Ameliorates Cartilage Degradation: Implications for the Treatment of Rheumatoid Arthritis.
ヒト・動物細胞 MH7A(RCB1512) Anti-angiogenic effect of Shikonin in rheumatoid arthritis by downregulating PI3K/AKT and MAPKs signaling pathways.
ヒト・動物細胞 MH7A(RCB1512) Near-infrared fluorescence imaging-guided focused ultrasound-mediated therapy against Rheumatoid Arthritis by MTX-ICG-loaded iRGD-modified echogenic liposomes.
ヒト・動物細胞 MH7A(RCB1512) Preparative separation of the flavonoid fractions from Periploca forrestii Schltr. ethanol extracts using macroporous resin combined with HPLC analysis and evaluation of their biological activities.
ヒト・動物細胞 CHO-K1 Effects of Soluble Tumor Necrosis Factor (TNF) on Antibody-Dependent Cellular Cytotoxicity of Therapeutic anti-TNF-α Antibody.
ヒト・動物細胞 MH7A(RCB1512) miR-708-5p promotes fibroblast-like synoviocytes' cell apoptosis and ameliorates rheumatoid arthritis by the inhibition of Wnt3a/β-catenin pathway.
ヒト・動物細胞 MH7A(RCB1512) MicroRNA-766-3p Contributes to Anti-Inflammatory Responses through the Indirect Inhibition of NF-κB Signaling.
ヒト・動物細胞 MH7A(RCB1512) Effects of Ghrelin on the Apoptosis of Rheumatoid Arthritis Fibroblast-Like Synoviocyte MH7A Cells.
ヒト・動物細胞 MH7A(RCB1512) S-propargyl-cysteine attenuates inflammatory response in rheumatoid arthritis by modulating the Nrf2-ARE signaling pathway.
実験動物マウス RBRC01390 Licochalcone A activates Keap1-Nrf2 signaling to suppress arthritis via phosphorylation of p62 at serine 349.
ヒト・動物細胞 MH7A(RCB1512) Anti-inflammatory and apoptotic effects of the polyphenol curcumin on human fibroblast-like synoviocytes.
ヒト・動物細胞 CCRF-HSB-2(RCB0016) Dipeptidyl peptidase IV on activated T cells as a target molecule for therapy of rheumatoid arthritis.